In Alzheimer’s disease, glutamate excitotoxicity which can damage neurons through chronic activation is exacerbated by deficiency of ABCA7, the second strongest genetic risk factor for the disease. The findings highlight ABCA7 and glutamate–GABA signaling as potential therapeutic targets.

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Hatch新收录的资料对此有专业解读

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搭载高通骁龙 7s Gen 4,搭配 UFS 3.1 存储;

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